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The sixC-terminal lysines of p53 are the predominant sites for mdm2-mediated ubiquitina-tion [76]. There wasno difference in the duration of symptoms of the common cold betweengroups.

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For example, embryonic lethalityof either MDM2 or MDMX-null mice can be completely rescued by the simultane-ous deletion of the TP53 gene. This is evidencethat Tregs are apart of a generalized system of immune control and that they have the abilityto induce homeostatic conditions within the entire immune system.

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